FASTKD2

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AliasesFASTKD2, KIAA0971, FAST kinase domains 2, COXPD44
End206,796,189 bp[1]
FASTKD2
Identifiers
AliasesFASTKD2, KIAA0971, FAST kinase domains 2, COXPD44
External IDsOMIM: 612322; MGI: 1922869; HomoloGene: 8957; GeneCards: FASTKD2; OMA:FASTKD2 - orthologs
Orthologs
SpeciesHumanMouse
Entrez
Ensembl
UniProt
RefSeq (mRNA)

NM_014929
NM_001136193
NM_001136194

NM_172422

RefSeq (protein)

NP_001129665
NP_001129666
NP_055744

NP_766010

Location (UCSC)Chr 2: 206.77 – 206.8 MbChr 1: 63.77 – 63.79 Mb
PubMed search[3][4]
Wikidata
View/Edit HumanView/Edit Mouse

FAST kinase domain-containing protein 2 (FASTKD2) is a protein that in humans is encoded by the FASTKD2 gene on chromosome 2.[5][6] This protein is part of the FASTKD family, which is known for regulating the energy balance of mitochondria under stress. FASTKD2 has been implicated in mitochondrial encephalomyopathy, breast cancer, and prostate cancer.[7][8][9]

FASTKD2 shares structural characteristics of the FASTKD family, including a ~50-amino acid N-terminal mitochondrial targeting domain and three C-terminal domains: two FAST kinase-like domains (FAST_1 and FAST_2) and a RNA-binding domain (RAP).[7][8] The mitochondrial targeting domain directs FASTKD2 to be imported into the mitochondria. Though the functions of the C-terminal domains are unknown, RAP possibly binds RNA during trans-splicing.[7]

Function

As a member of the FASTKD family, FASTKD2 localizes to the inner mitochondrial membrane to modulate their energy balance, especially under conditions of stress.[7][8] Though ubiquitously expressed in all tissues, FASTKD2 appears more abundantly in skeletal muscle, heart muscle, and other tissues enriched in mitochondria.[7] Nonetheless, FASTKD2 has been observed to mediate apoptosis independent of import into the mitochondria, suggesting that it interacts with proteins on the outer mitochondrial membrane. This protein possibly contributes its proapoptotic function through a caspase-dependent pathway, by activating proapoptotic factors or inhibiting antiapoptotic factors, but the exact mechanism remain unclear.[8][9] FASTKD2 has also been validated as an RNA-binding protein.[10][11]

Clinical significance

Interactions

References

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